Paediatric ECG Basics
Appcardio ECG Academy
Paediatric ECG Basics
A practical, age-aware approach to acquiring and interpreting the paediatric electrocardiogram—from normal developmental patterns to common rhythm and repolarisation abnormalities.
Learning objectives
1. Why the paediatric ECG is different
A child's ECG is not simply a smaller adult ECG. During the transition from fetal to postnatal circulation, pulmonary vascular resistance falls and the relative dominance of the right ventricle gradually gives way to a more balanced—and eventually left-ventricular-dominant—pattern. Heart rate, electrical axis, QRS voltages, repolarisation and conduction intervals therefore vary with age.
Expected developmental patterns
- Heart rate is generally faster in infants and young children than in adults.
- Right ventricular forces are prominent in the newborn; the QRS axis is often rightward and the right precordial R wave may be relatively tall.
- PR and QRS durations are generally shorter in younger children.
- Right precordial T-wave inversion is a common developmental pattern after the first few days of life and may persist into adolescence.
- Voltage criteria for chamber enlargement must be judged against age-specific standards; isolated high voltage is not sufficient to diagnose hypertrophy.
2. ECG acquisition and lead placement
Before interpreting morphology, confirm that the tracing is technically reliable. A misplaced electrode can produce apparent axis, bundle-branch or repolarisation abnormalities.
Technical checklist
- Confirm the child's identity, age and date/time of recording.
- Check calibration and paper speed. Standard settings are usually 25 mm/s and 10 mm/mV.
- Ensure the child is warm, as still and comfortable as possible; reduce movement and muscle artefact.
- Use appropriately sized electrodes and ensure good skin contact.
- Confirm limb and precordial electrode positions before accepting an unexpected pattern.
Standard precordial landmarks
- V1: fourth intercostal space, right sternal border.
- V2: fourth intercostal space, left sternal border.
- V3: midway between V2 and V4.
- V4: fifth intercostal space, midclavicular line.
- V5: level with V4, anterior axillary line.
- V6: level with V4, midaxillary line.
| Lead group | Placement | Practical note |
|---|---|---|
| Limb electrodes | Right arm, left arm, right leg and left leg | Use the standard limb positions; keep limb labels consistent and avoid swapping right and left. |
| V1–V2 | Fourth intercostal spaces at the sternal borders | High placement can alter P-wave and QRS morphology, particularly in V1–V2. |
| V3–V6 | Follow the anatomical landmarks listed above | V4 should be positioned before V3; V5 and V6 remain horizontally level with V4. |
| Additional right-sided leads | V4R is the mirror position of V4 on the right chest | May be useful when right ventricular pathology is suspected; document additional leads clearly. |
Illustration: original Appcardio schematic for orientation; it is not a substitute for palpating anatomical landmarks.
3. A stepwise approach to interpretation
Use the same disciplined sequence for every tracing. Automated measurements can support interpretation but should not replace visual review and manual verification.
- Patient and technical details. Verify identity, age, clinical indication, paper speed, calibration, artefact and lead placement.
- Rate. Calculate atrial and ventricular rates when they differ. Decide whether the rate is appropriate for age and clinical state.
- Rhythm. Assess regularity, P-wave presence and morphology, the relationship between P waves and QRS complexes, and PR consistency.
- Axis. Determine the QRS axis and compare it with the expected range for age.
- Intervals. Measure PR, QRS and QT; calculate QTc using an appropriate correction method and interpret in context.
- Waveform morphology. Review P-wave morphology, Q waves, R/S progression, voltages and evidence of chamber enlargement or conduction abnormality.
- ST segments and T waves. Look for repolarisation changes, remembering normal age-related T-wave patterns.
- Overall synthesis. State whether the ECG is normal or abnormal for age, identify key abnormalities and relate them to the clinical question.
Paper speed and calibration
| Measurement | At 25 mm/s | At 10 mm/mV |
|---|---|---|
| One small horizontal square (1 mm) | 0.04 seconds (40 ms) | — |
| One large horizontal square (5 mm) | 0.20 seconds (200 ms) | — |
| Vertical deflection of 1 mm | — | 0.1 mV |
| Vertical deflection of 10 mm | — | 1 mV |
Rate estimate: for a regular rhythm at 25 mm/s, heart rate ≈ 300 divided by the number of large squares between consecutive R waves, or 1,500 divided by the number of small squares. For an irregular rhythm, count QRS complexes over a known interval (for example, 10 seconds × 6).
4. Normal values change with age
The values below are selected reference data from the Royal Children's Hospital Melbourne paediatric ECG guideline. They are guides rather than universal cut-offs: reference ranges vary by population, age grouping, heart rate and measurement method.
Heart rate and QRS axis
| Age | Mean heart rate (bpm) | Heart-rate range (bpm) | Mean QRS axis | QRS-axis range |
|---|---|---|---|---|
| Newborn | 145 | 120–170 | — | — |
| 6 months | 145 | 110–170 | — | — |
| 1 year | 132 | 105–150 | — | — |
| 2 years | 120 | 95–150 | +60° | +10° to +110° |
| 4 years | 108 | 80–150 | +60° | +10° to +110° |
| 6 years | 100 | 75–140 | +60° | +20° to +120° |
| 10 years | 90 | 60–130 | +60° | +20° to +120° |
| 14 years and older | 85 | 60–115 | +60° | +20° to +120° |
The RCH guideline separately reports QRS axis of +110° (range +30° to +180°) from 1 week to 1 month, and +70° (range +10° to +125°) from 1 to 3 months. Its reference for children older than 3 years is +60° (range +20° to +120°); adult limits are listed separately. Dashes above mean the selected age-specific table did not provide a corresponding value at that row.
Conduction intervals: upper limits of normal
| Age band | PR upper limit (ms)* | QRS upper limit (ms) |
|---|---|---|
| 0–1 month | Up to 120–140, depending on rate | 70 |
| 1–6 months | Up to 130–140, depending on rate | 75 |
| 6–12 months | Up to 130–140, depending on rate | 75 |
| 1–3 years | Up to 140, depending on rate | 75 |
| 3–8 years | Up to 150–170, depending on rate | 75 |
| 8–12 years | Up to 150–180, depending on rate | 85 |
| 12–16 years | Up to 160–190, depending on rate | 85 |
*PR upper limits are heart-rate dependent in the RCH reference table; the ranges shown are a concise summary of the tabulated upper limits, not a single threshold for all rates. Consult the full age- and rate-stratified table when borderline measurements matter. RCH QRS upper limits are based on the 98th percentile.
5. Recognising normal paediatric patterns
Sinus rhythm and sinus arrhythmia
Sinus rhythm is supported by a consistent P wave before each QRS, a stable PR relationship, and a P-wave axis compatible with sinus origin (upright in leads I and aVF in the RCH guideline). Respiratory sinus arrhythmia—gradual variation in R–R intervals with breathing—is common in children and is usually physiological.
Right ventricular dominance in early life
Right-sided forces are expected to be more prominent in neonates and young infants. With growth, the QRS axis and precordial R/S balance evolve. Do not apply adult voltage or axis expectations to a newborn.
Juvenile T-wave pattern
T waves are often inverted in V1–V3 during childhood. They generally become upright progressively during adolescence, but persistent inversion in V1 can remain a normal variant. T waves in V5 and V6 should ordinarily be upright.
ST segments
Small ST shifts can occur in children: the RCH guideline notes that up to 1 mm in limb leads and up to 2 mm in precordial leads may be normal. Persistent horizontal ST depression, marked elevation or changes that fit the clinical picture require assessment rather than dismissal as a variant.
6. Common paediatric arrhythmias
The supplied LITFL lesson identifies common paediatric arrhythmias as a core topic but does not provide diagnostic criteria in the supplied PDF extract. The following is a concise supplementary teaching overview; rhythm diagnosis should be made from the complete tracing and clinical state.
| Rhythm | ECG pattern to assess | Clinical interpretation |
|---|---|---|
| Sinus tachycardia | Sinus P waves before each QRS; rate varies with physiological demand and may show gradual onset/offset. | Look for causes such as fever, pain, dehydration, hypovolaemia, sepsis, hypoxia or medication. |
| Supraventricular tachycardia (SVT) | Often a very regular narrow-complex tachycardia; P waves may be absent, hidden or retrograde. Rate alone is not diagnostic. | Assess perfusion and haemodynamic stability immediately; obtain expert paediatric support. |
| Ventricular tachycardia | Usually a broad-complex tachycardia; AV dissociation, capture or fusion beats may support the diagnosis when present. | Potentially life-threatening. Treat an undifferentiated broad-complex tachycardia as VT until expert assessment indicates otherwise. |
| Atrial flutter / atrial fibrillation | Look for organised flutter activity or an irregularly irregular ventricular response without consistent P waves. | Uncommon in otherwise healthy children; consider underlying cardiac disease, surgery or systemic illness. |
| Bradycardia / AV block | Assess P-wave rate, QRS rate, PR behaviour and whether atrial impulses conduct to the ventricles. | Correlate with age, sleep, medication, hypoxia and signs of raised intracranial pressure or conduction disease. |
7. Other ECG abnormalities in children
The supplied LITFL source specifically lists the following additional abnormalities as relevant paediatric ECG topics:
Inflammatory and myocardial conditions
- Pericarditis: often viral in children; consider diffuse ST–T changes in the appropriate clinical setting.
- Myocarditis: may produce non-specific ST–T changes, low voltage, conduction disturbance or arrhythmia; ECG can also be non-diagnostic.
Electrolyte-related changes
- Hypokalaemia: may cause T-wave flattening, ST depression and prominent U waves.
- Hyperkalaemia: may cause peaked T waves, PR prolongation, QRS widening and, in severe cases, loss of visible P waves.
- Hypocalcaemia: typically prolongs the QT interval through ST-segment prolongation.
- Hypercalcaemia: typically shortens the QT interval.
These are pattern-recognition prompts, not stand-alone diagnostic criteria. Confirm suspected electrolyte abnormalities with laboratory testing and interpret ECG changes in clinical context.
8. Red flags and interpretation pitfalls
Clinical red flags
- Exertional syncope or exertional chest pain.
- Palpitations associated with syncope, near-syncope or haemodynamic compromise.
- Reduced exercise tolerance or unexplained breathlessness.
- Family history of premature sudden cardiac death, inherited arrhythmia or cardiomyopathy.
- Abnormal cardiac examination or known congenital/acquired heart disease.
Common interpretation errors
- Using adult reference values in infants and children.
- Accepting automated axis, QT or interval measurements without checking the tracing.
- Misplacing V1–V2 or swapping limb leads.
- Calling right ventricular hypertrophy from voltage alone.
- Labelling a juvenile T-wave pattern as ischaemia without considering age and distribution.
- Reassuring solely on the basis of a normal ECG when the history contains cardiac red flags.
9. Knowledge check
Test the key principles from this lesson. Select an answer and check the explanation.
10. Key takeaways
- Paediatric ECGs change substantially with age; they must not be interpreted using adult norms alone.
- Check identity, calibration, paper speed and electrode placement before interpreting the tracing.
- Follow a reproducible sequence: rate, rhythm, axis, intervals, morphology, ST segments and T waves.
- Right ventricular dominance in early life and juvenile T-wave inversion can be normal developmental findings.
- Interpret the ECG alongside the history and examination. A normal tracing does not exclude important cardiac disease in a child with red flags.
References
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